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Tinalyn Kupfer, PhD

Research Associate CCTCARE and Gill Lab

Dr. Kupfer is a basic scientist, clinical researcher and scientific/medical writer.  She is described as a cross-functional scientist in translational/clinical research.  In 1996 she completed her BS in Biology at the University of New Mexico, Albuquerque and followed up with her PhD in Immunology at the University of Colorado Health Sciences Center in 2003. 

She supervises the technical protocols/standard operating procedures (SOPs) of the multiple lab groups with the CCTCARE group.  Additional duties include organization and oversight of regulated activities such as the use of biohazardous, radioactive, or human blood/tissue used as part of the research actitivies.

Phone: 303.724.5469
Fax: 303.724.7624
Email: tinalyn.kupfer@ucdenver.edu

Kupfer TM, Pham K, Beilke JN, and Gill RG. 2006. Acute destruction of islet allografts in NOD mice is donor MHC-independent. American Journal of Transplantation. 6 (S2):2860.

Kupfer TM and Gill RG. 2006. NOD antigen-presenting cells are sufficient to render islets vulnerable to autoimmune injury in an MHC-disparate environment. American Journal of Transplantation. 6 (S2):2092.

Kupfer TM, Crawford ML, and Gill RG. 2006 IFN-g , but not TNFR1 or IL-1R, is a key component of recurrent autoimmune pathogenesis to islet allografts mediated by indirect, islet-autoreactive CD4 T cells. Diabetes. 55 (S1):97-OR.

Kupfer TM, Crawford ML, and Gill RG. 2005. IFN-g is a key component of CD4 T cell-mediated recurrent autoimmune pathogenesis to islet allografts. American Journal of Transplantation. 5 (S11): 309.

Kupfer TM, Crawford ML, Pham K, and Gill RG. 2005. MHC-mismatched islet allografts are vulnerable to autoimmune recognition in vivo. Journal of Immunology. 175 (4): 2309-2316.

Malecki TM, Rayat GR, and Gill RG. 2002. Autoreactive, islet-specific mouse CD4 T cells coordinate the destruction of islet xenografts in vivo. American Journal of Transplantation. 2 (S3):1302.

Malecki TM, Crawford ML, and Gill RG. 2001. Islet allograft destruction by islet-specific CD4 T cells occurs via indirect antigen presentation. Keystone Symposia on Tolerance and Autoimmunity.

Malecki TM, Crawford ML, and Gill RG. 2000. Islet allograft destruction by islet-specific CD4 T cells occurs via indirect antigen presentation. Transplantation Vol. 69 (8) Suppl.

Malecki TM, Jillson GP, Thilsted JP, Elrod J, Torrez-Martinez N, and Hjelle B. 1998. Serologic survey for Hantavirus infection in domestic animals and coyotes from New Mexico and northeastern Arizona. Journal of the American Veterinary Medical Association. 2312:970-973.

Malecki TM, Baur KL, and Goldberg EH. 1996. In vitro proliferation of lymph node and spleen cells from (B6xA)/B6 radiation chimeras to Skn alloantigens. FASEB