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Dr. Wang joined the Division of Endocrinology in 2006. She received her Bachelor of Science degree in chemistry from Nanjing University, Nanjing, China in 1990. She then came to United States to pursue graduate study and received her Ph.D in Biochemistry from University of Utah, Salt Lake City, Utah in 1998. She went on to get postdoctoral training in Huntsman Cancer Institute at University of Utah, where she researched in the area of transcriptional regulation mechanism and cancer biology. Before coming to CU Denver, she was the Director of Protein Expression and Purification Facility at Colorado State University. Dr. Wang currently is the manager for the research group of Dr. Robert Eckel.

Selected Publications 

  • Maahs DM, Hokanson JE, Wang H, Kinney GL, Snell-Bergeon JK, East A, Bergman B, Schauer IE, Rewers M, Eckel RH. ”Lipoprotein Subfraction Cholesterol Distribution is Pro-Atherogenic in Women with Type 1 Diabetes and Insulin Resistance.” Diabetes. 2010 Apr 14. [Epub ahead of print]
  • Wang H, Eckel RH Lipoprotein lipase: from gene to obesity. Am J Physiol Endocrinol Metab. 2009 Aug;297(2):E271-88. PUBMED
  • Wang H, Knaub LA, Jensen DR, Young Jung D, Hong EG, Ko HJ, Coates AM, Goldberg IJ, de la Houssaye BA, Janssen RC, McCurdy CE, Rahman SM, Soo Choi C, Shulman GI, Kim JK, Friedman JE, Eckel RH.Skeletal muscle-specific deletion of lipoprotein lipase enhances insulin signaling in skeletal muscle but causes insulin resistance in liver and other tissues. Diabetes. 2009 Jan;58(1):116-24. PUBMED
  • Cornier MA, Dabelea D, Hernandez TL, Lindstrom RC, Steig AJ, Stob NR, Van Pelt RE, Wang H, Eckel RH. The metabolic syndrome. Endocr Rev. 2008 Dec;29(7):777-822. PUBMED
  • Wang, H; McIntosh, L.P.; Graves, B.J. “Inhibitory module of Ets-1 allosterically regulates DNA binding through a dipole-facilitated phosphate contact.” J. Biol. Chem., Vol. 277(3), 2225-2233, 2002


Coming Soon!

Reasearch Interests

The main focus of Dr. Wang’s research is to study the role of lipoprotein lipase in the metabolic processing of triglyceride-rich lipoproteins in central nerve system, skeletal muscles as well as in other organs in the body using unique genetically-modified mouse models. The goal is to understand how the modifications in the expression and regulation of LPL lead to unbalanced nutrient partition that has been implicated in several disease states including obesity, diabetes mellitus, dyslipidemias, and associated cardovascular disorders.


  • Lipid Metabolism